Am J Clin Nutr：胖子餐后合成代谢敏感性更低

2016-09-13 Mechront 译 MedSci原创

在对高胰岛素血症-高氨基酸血症钳夹试验反应过程中，过量的身体脂肪会减少肌肉蛋白质合成率。我们对肥胖、超重与正常体重成年人进行了调查研究，比较他们在摄入瘦肉之后的肌原纤维蛋白合成反应和潜在的营养感应机制。肥胖(OB; BMI: 35.9 ± 1.3)、超重（OW; BMI: 27.1 ± 0.5）与正常体重（HW; BMI 22.7 ± 0.4）成年人各有10例，接受引物连续l-[ring

在对高胰岛素血症-高氨基酸血症钳夹试验反应过程中，过量的身体脂肪会减少肌肉蛋白质合成率。我们对肥胖、超重与正常体重成年人进行了调查研究，比较他们在摄入瘦肉之后的肌原纤维蛋白合成反应和潜在的营养感应机制。

肥胖(OB; BMI: 35.9 ± 1.3)、超重（OW; BMI: 27.1 ± 0.5）与正常体重（HW; BMI 22.7 ± 0.4）成年人各有10例，接受引物连续l-[ring-13C6]苯丙氨酸注射。在摄入170 g猪肉（36克蛋白质和3克脂肪）前后，收集参与者的血液和肌肉样本，评估骨骼肌代谢、氨基酸转运载体（大中性和小中性氨基酸转运蛋白(LAT1, SNAT2)和CD98）、以及肌原纤维蛋白的合成。

在基线，相比HW组，OW和OB组哺乳动物雷帕霉素靶物1（mTORC1）蛋白的相对含量更高。摄入猪肉后，只有HW组的mTORC1磷酸化增加(P = 0.001)。所有组参与者摄入食物后，其LAT1和SNAT2蛋白含量增加（时间效应，P＜0.05）。组间基底肌原纤维蛋白合成反应相似（P = 0.43）。摄入食物后，相比OW和OB组，HW组参与者的肌原纤维蛋白质合成反应（0–300 min）更大（1.6倍；P = 0.005）。

结果表明，与正常体重对照组相比，超重和肥胖成年人摄入蛋白质丰富的食物后，其肌原纤维蛋白合成反应减少了。这些数据表明，受损后的肌原纤维蛋白合成反应，可能是随着脂肪量增加的早期表现。

原始出处：

Joseph W Beals.et al.Anabolic sensitivity of postprandial muscle protein synthesis to the ingestion of a protein-dense food is reduced in overweight and obese young adults.Am J Clin Nutr ajcn130385; First published online September 7, 2016

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2016-09-22 1e10c84am36(暂无匿称)

文章很好，非常有益

94 0
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2016-09-16 lhl629

学习了，很好。

71 0
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2016-09-15 舒心和人

很有道理，感谢分享

78 0
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2016-09-15 ms5236388012055669

#敏感性#

53 0
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2016-09-14 1dd8a7c5m95(暂无匿称)

学习了，赞一个！！！

76 0
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2016-09-14 1dd8a7c5m95(暂无匿称)

学习了，赞一个！！！

98 0
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2016-09-13 doctorJiangchao

继续学习

49 0
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