Diabetologia：某维生素D基因+高蛋白饮食=改善血糖

2015-10-13 Mechront 译 MedSci原创

维生素D和相关遗传变异与肥胖和胰岛素抵抗有关。研究者进行了一项研究，探究维生素D代谢相关基因变异是否会对体重和胰岛素抵抗产生影响，以应对不同营养素为主的减肥饮食。一项对732名超重/肥胖研究对象长达2年的减重试验(POUNDS Lost)发现了3种维生素D代谢相关基因变异：DHCR7 rs12785878；CYP2R1 rs10741657；GC rs2282679 。研究者评估了不同基因型对以下

维生素D和相关遗传变异与肥胖和胰岛素抵抗有关。研究者进行了一项研究，探究维生素D代谢相关基因变异是否会对体重和胰岛素抵抗产生影响，以应对不同营养素为主的减肥饮食。

一项对732名超重/肥胖研究对象长达2年的减重试验(POUNDS Lost)发现了3种维生素D代谢相关基因变异：DHCR7 rs12785878；CYP2R1 rs10741657；GC rs2282679 。研究者评估了不同基因型对以下方面的影响：体重变化、空腹血糖和胰岛素水平、第6个月（656研究对象）和2年（595 研究对象）稳态胰岛素评价指数（HOMA-IR）。

研究结果显示，研究者发现DHCR7 rs12785878和不同蛋白的饮食具有明显的交互作用，不过对于脂类则没有发现该联系；并且还发现了其对胰岛素、6个月时HOMA-IR (p for interaction <0.001)、2年时HOMA-IR (p for interaction ≤0.03)的交互作用。DHCR7 rs12785878的T等位基因（增加 VD的等位基因）在高蛋白饮食中，可显著降低胰岛素水平和HOMA-IR (p < 0.002)；不过对于低蛋白饮食没有发现该基因型对这些检测指标的影响。广义估计方程分析表明，超过2年的高蛋白饮食干预，该基因型可显著影响胰岛素抵抗的轨迹变化(p < 0.001)。研究者没有观测到其他两个基因型在蛋白或脂类饮食中的交互作用。

研究结果表明，携带HCR7 rs12785878基因的T等位基因人群，在食用高蛋白减肥饮食时，比没有携带该等位基因的人群，对胰岛素抵抗的改善情况更受益，即是说可以更好的控制血糖。

原始出处：

Qibin Qi, Yan Zheng.et al.Vitamin D metabolism-related genetic variants, dietary protein intake and improvement of insulin resistance in a 2 year weight-loss trial: POUNDS Lost.Diabetologia.September 29, 2015.

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值得学习

194 0
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2016-04-10 忠诚向上

值得学习

172 0
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2016-04-10 忠诚向上

值得学习

187 0
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2016-04-10 忠诚向上

值得学习

148 0
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2016-04-10 忠诚向上

值得学习

186 0
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2016-04-10 忠诚向上

值得学习

109 0
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2016-04-10 忠诚向上

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