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SCI REP：原发性局灶节段性肾小球硬化患者血清C3与肾脏结局的关系！

2017-06-22 xing.T MedSci原创

FSGSC3肾脏结局
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总之，FSGS患者发生的补体活化与临床和组织学严重程度相关。低血清C3水平是FSGS患者不良预后的独立危险因素。

补体（C）在局灶节段性肾小球硬化（FSGS）的发病机制或进展中的作用是不确定的。近日，Scientific reports 杂志上发表了一篇研究文章，研究人员旨在评估了队列中血清C3水平与FSGS患者基础特征和FSGS进展之间的相关性，以及明确FSGS患者血清补体C3水平的临床意义。

研究人员发现相比于血清C3≥85毫克/分升（n=474）的参与者，那些C3<85 毫克/分升（n=117）的参与者呈现出较高的血清肌酐水平，较低的EGFR、血红蛋白、尿蛋白、甘油三酯、胆固醇和IgA水平，以及重度肾小管间质损伤（TI）。

研究人员对221例患者平均随访53.3个月，在血清补体C3低水平患者中达到终末期肾脏疾病（ESRD）的风险显著较高（P＜0.001）。额外的40例原发性FSGS患者研究结果显示MAC和AP之间（P=0.003）、MAC和血清C3之间（P=0.018）以及AP和血清C3之间（P=0.028）存在显著的相关性。与无至轻度TI患者相比，中度至重度TI患者血清C3和AP水平较低，但血清MAC水平更高。

总之，FSGS患者发生的补体活化与临床和组织学严重程度相关。低血清C3水平是FSGS患者不良预后的独立危险因素。

原始出处：

Jian Liu,et al. Serum C3 and Renal Outcome in Patients with Primary Focal Segmental Glomerulosclerosis.Scientific reports. 2017. https://www.nature.com/articles/s41598-017-03344-1

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    2021-06-26 为了健康

    学习了

    0

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    2017-10-19 jiyangfei

    #原发性#

    0

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    2017-06-24 szhvet

    #肾脏结局#

    0

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    2017-06-23 1e1c07bdm01(暂无匿称)

    好

    0

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    2017-06-22 中医痴

    不错的，学习了，谢谢分享！

    0

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    2017-06-22 有备才能无患

    FSGS患者发生的补体活化与临床和组织学严重程度相关。低血清C3水平是FSGS患者不良预后的独立危险因素。

    0

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